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Treg cells are essential for the autoimmune pathogenesis of GN in the kidney (353), as such, the activation of STAT3 in Treg cells induces the expression of CC chemokine receptor 6 (CCR6) on the cell surface (354) and promotes the transport of Treg cells to the inflammatory area of Th17 cells that also highly express CCR6 through the tight colocalization of CCR6 (355), thereby suppressing the immune response of pathogenic Th17 cells during the GN process

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